Vaughan Williams Classification (03:04)
Antiarrhythmic agents are categorized according to how they affect cardiac cell action potential. View indications, functions, and drugs for classes I, II, III, and IV. Adenosine is an A1 receptor agonist; digoxin increases vagal activity.
Class I Antiarrhythmic Agents (04:36)
Membrane stabilizing agents affect action potential phases 0 and 1. They include: quinidine, procainamide, disopyramide, and lidocaine. Learn about indications, administration route, dosage, and side effects for each. They can have a "proarrhythmic" effect increasing ventricular tachycardia; continuous ECG monitoring is required.
Class II Antiarrhythmic Agents (02:37)
Beta blockers depress phase 4 depolarization, suppress automaticity and slow AV nodal conduction by reducing sympathetic nervous system stimulation to the heart, resulting in decreased impulse transmission. Learn about indications, administration routes, and contraindications for metoprolol and atenolol.
Class III Antiarrhythmic Agents (03:11)
Class III agents prolong repolarization during action potential phase 3 and inhibit adrenergic stimulation by blockading potassium channels. They include sotalol, amiodarone, dofetilide, and dronedarone. Learn about indications, administration route, side effects, and nursing assessments.
Class IV Antiarrhythmic Agents (02:05)
Calcium channel blockers include verapamil and diltiazem, and are indicated for paroxysmal supraventricular tachycardia, atrial fibrillation, and atrial flutter. Learn about administration routes and contraindications.
View a scenario in which medical professionals assess a supraventricular tachycardia patient. Chemically unrelated to antiarrhythmic drugs, adenosine slows conduction time through the AV node through parasympathetic system stimulation. Learn about administration route, dosage, and immediate side effects.
Credits: Antiarrhythmic Agents 2 (00:13)
Credits: Antiarrhythmic Agents 2
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